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These chemicals are generated from injured or stressed cells in response to ROS formation and operate as signaling factors to recruit and activate immune cells, sustaining inflammation (Albano et al., 2022)

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However, one study found that certain tumor cells were still able to survive and proliferate after GPX4 deletion

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Figure 2E] was significantly higher than the placebo group at baseline

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Glutathione peroxidase 4 overexpression inhibits ROS-induced cell death in diffuse large B-cell lymphoma

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